Formation of the androgen receptor transcription complex.

By Yongfeng Shang, M. Myers, Myles A. Brown. Molecules and Cells, 2002

705 citations recorded by the source corpus.

Subfields: Genetics, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/653995f8d58662a8278917aba4a0a61d4c79ca07

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Genetics

1. C. Sawyers
2. Jason S. Carroll
3. Myles A. Brown
4. Arul M. Chinnaiyan
5. X. S. Liu
6. Wei Li
7. Rory Stark

In Pulmonary and Respiratory Medicine

1. Arul M. Chinnaiyan
2. Mark A. Rubin
3. R. Vessella
4. C. Sawyers

Citations in the corpus

• Androgen-Induced Differentiation and Tumorigenicity of Human Prostate Epithelial Cells (2004, neutral): “the pWZL retrovirus with a blasticidin selection cassette (22).”
• Ligand-independent Androgen Receptor Variants Derived from Splicing of Cryptic Exons Signify Hormone Refractory Prostate Cancer (2009, neutral): “Androgen binding to the AR LBD allows entry of the ligand-bound receptor into the nucleus and subsequent transcriptional regulation of androgen-responsive genes (3).”
• A hierarchical network of transcription factors governs androgen receptor-dependent prostate cancer growth. (2007, background): “Selected AR Binding Sites Play Transcriptional Roles We and others have previously found that RNA Pol II associates with the AR binding regions of the PSA enhancer and promoter, which is critical for PSA transcription (Louie et al., 2003; Shang et al., 2002; Wang et al., 2005).”
• Androgen Receptor Regulates a Distinct Transcription Program in Androgen-Independent Prostate Cancer (2009, background): “…138, 245–256, July 24, 2009 ª2009 Elsevier Inc. 245 that control of these target genes involves long-range, combinatorial regulation by AR, DNA-binding collaborating transcription factors and non-DNA binding co-regulatory factors (Shang et al., 2002; Wang et al., 2005; Wang et al., 2007).”
• Molecular determinants of resistance to antiandrogen therapy (2004, background): “, and that androgen induces growth arrest or apoptosis in certain context”
• Dual role of FoxA1 in androgen receptor binding to chromatin, androgen signalling and prostate cancer (2011, background): “To form a productive transcription complex on chromatin and to bring about diverse biological actions of androgens, AR needs to communicate with coregulatory proteins (coactivators and corepressors) and collaborating transcription factors (Shang et al, 2002; Kang et al, 2004; Wang et al, 2005).”