Causal models of attention-deficit/hyperactivity disorder: from common simple deficits to multiple developmental pathways.

By Edmund Sonuga-Barke. Biological Psychiatry, 2005

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Normal Development of Brain Circuits (2010, background): “Sonuga-Barke E (2005). Causal models of attention-deficit/hyperactivity disorder: from common simple deficits to multiple developmental pathways.”
Reward anticipation and outcomes in adult males with attention-deficit/hyperactivity disorder (2008, background): “Delay aversion in ADHD was also associated with sensation seeking behavior, impulsivity and hyperactivity as a compensation strategy in situations with poor stimulation ( Sonuga-Barke, 2002, 2005 ).”
Spontaneous attentional fluctuations in impaired states and pathological conditions: a neurobiological hypothesis. (2007, background): “Recent models have emphasized this psychopathophysiological heterogeneity (Sonuga-Barke, 2005) and developed the idea that ADHD is an umbrella construct, which while clinically useful, subsumes multiple groups of patients with distinctive aetiological and pathophysiological profiles.”
FROM REINFORCEMENT LEARNING MODELS OF THE BASAL GANGLIA TO THE PATHOPHYSIOLOGY OF PSYCHIATRIC AND NEUROLOGICAL DISORDERS (2011, background): “'Multiple-pathway' accounts suggest that executive dysfunction and excessive delay discounting are both involve”
Changes and challenges in 20 years of research into the development of executive functions (2011, background): “As a result, contemporary causal accounts of ADHD (e.g., Sonuga-Barke, 2005) also include additional deficits in the signaling of delayed rewards.”