Advances in metal-induced oxidative stress and human disease.
By K. Jomová, M. Valko. Toxicology, 2011
3,025 citations recorded by the source corpus.
Subfields: Health, Toxicology and Mutagenesis, Nutrition and Dietetics
View Paper: https://www.semanticscholar.org/paper/6429357797b1faec9700244a00e58ebd0402597e
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In Health, Toxicology and Mutagenesis
In Nutrition and Dietetics
Citations in the corpus
- Zinc and human health: an update (2011, neutral): “Zinc deficiency, after prolonged reduction of intake or excessive uncompensated losses, has been described both in animals and humans and is associated with increased levels of oxidative damage, including increased lipid, protein, and DNA oxidation (Prasad 2009; Jomovaa and Valko 2011).”
- The Role of Oxidative Stress in Parkinson’s Disease (2013, background): “Iron ions can generate ROS since ferric iron (Fe 3+ ) and ferrous iron (Fe 2+ ) can react with superoxide and hydrogen peroxide, respectively, in a chain reaction generating the highly reactive hydroxyl free radical, which together with dopamine oxidation can trigger neurotoxicity [109] [110].”
- Redox regulation of FoxO transcription factors (2015, background): “Exposure of cells to arsenite – another molecule with high affinity towards thiols – results in oxidative damage to biomolecules [85,86], suggesting the generation of ROS.”
- The effects of heavy metals on human metabolism (2019, background): “…of the near-curved tube, isolates and releases free cadmium, thus interfering with the metabolite of the chowtoral epithelial cell mitochondrial energy metabolism and oxidation phosphorylation, which in turn leads to renal cell damage and death (Ohta and Cherian 1991; Jomova and Valko 2011).”
- Non-transferrin bound iron: a key role in iron overload and iron toxicity. (2012, neutral): “Iron is known to promote ROS via the Haber–Weiss reaction [113].”
- Copper: Toxicological relevance and mechanisms (2014, neutral): “The underlying mechanism of action for transition metal ions may involve the formation of superoxide, hydroxyl radical, and other ROS and subsequently producing malondialdehyde, 4-hydrox- ynonenal, and other exocyclic DNA adducts (Rossi et al. 2006; Gaetke and Chow 2003; Jomova and Valko 2011).”
- Duckweeds for water remediation and toxicity testing (2016, neutral): “Growing on and elicit destructive oxidative action on proteins, DNA, and lipids (Jomova and Valko 2011; Hossain et al. 2012).”
- Vapors Produced by Electronic Cigarettes and E-Juices with Flavorings Induce Toxicity, Oxidative Stress, and Inflammatory Response in Lung Epithelial Cells and in Mouse Lung (2015, background): “Heavy metals may undergo redox cycling and alter the oxidation state of the cell by potentiating the production of ROS [17].”
- Lipid Peroxidation: Production, Metabolism, and Signaling Mechanisms of Malondialdehyde and 4-Hydroxy-2-Nonenal (2014, background): “[406] K.”
- Familial Occurrence of KienbockÂ’s Disease (2011, background): “Besides the encymes (SOD, catalase), many low-molecular weight antioxidants (ascorbic acid, alpha-tocopherol, glutathione, carotenoids, flavonoids, and other antioxidants) are capable of chelating metal ions reducing their catalytic activity to form reactive oxygen species [173].”