Hyperglycemia-induced apoptosis in mouse myocardium: mitochondrial cytochrome C-mediated caspase-3 activation pathway.

By L. Cai, Wei Li, Guang-wei Wang, Luping Guo, Youchun Jiang, Y. J. Kang. Diabetes, 2002

702 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Epidemiology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/6361adbcd1f834c4821ab0d40753dad4ece26412

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Citations in the corpus

• The role of oxidative stress in diabetic complications (2007, neutral): “and cardiac myocytes (370) exposed to hyper-”
• Molecular mechanisms of diabetic cardiomyopathy (2014, neutral): “Increased cell death is frequently observed in hearts of humans with type 1 and type 2 diabetes and rodent models of these disorders, and includes both apoptotic and necrotic cell death [40–44].”
• Diabetic cardiomyopathy: evidence, mechanisms, and therapeutic implications. (2004, background): “Evidence in vivo has shown that hyperglycemia directly induces apoptotic cell death and myocyte necrosis in the myocardium, triggered by reactive oxygen species derived from high levels of glucose (122).”