X-inactivation, imprinting, and long noncoding RNAs in health and disease.
By Jeannie T. Lee, M. Bartolomei. Cell, 2013
652 citations recorded by the source corpus.
Subfields: Genetics, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/612eac6396877419b851fe6826256c5734caf0bd
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Citations in the corpus
- Long Noncoding RNA MALAT1 Regulates Endothelial Cell Function and Vessel Growth (2014, background): “inactivation.(8) NATs often regulate the associated sense transcript.”
- Long noncoding RNAs: Cellular address codes in development and disease (2013, neutral): “Some of the first studied examples of lncRNA function involve dosage compensation and genomic imprinting, whereby lncRNAs provide allele-specific gene regulation to differentially control two copies of the same gene within one cell (see the Review by Lee and Bartolomei on page 1308 of this issue; Le”
- Changes in sex ratio from fertilization to birth in assisted-reproductive-treatment cycles (2014, background): “Two recent reviews by Lee and Bartolomei [39] and Lessing et al.”
- Non-coding RNAs in Development and Disease: Background, Mechanisms, and Therapeutic Approaches. (2016, background): “For instance, IL1 -eRNA and IL1 RBT46 eRNAs attenuate LPS-induced mRNA transcription in human innate immune responses (117); Xist regulates genomic imprinting in cis (153), whereas HOTAIR is a trans-acting regulator of the HOXD genes (228).”
- Transcriptional and Post-transcriptional Gene Regulation by Long Non-coding RNA (2017, background): “In both cases, the lncRNA is expressed from the inactivated chromosome and has a binary relationship with its coding gene expression from the other chromosome, which is controlled by a cis-acting master control region [75].”