Chemokine: receptor structure, interactions, and antagonism.

By S. Allen, Susan E. Crown, Tracy M. Handel. Annual Review of Immunology, 2007

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Inflammation: gearing the journey to cancer. (2008, background): “, CXC, CC, XC and CX3C primarily based on the positions of conserved cysteine residues [1,61,109].”
Biology and Diseases of Mice (2015, positive): “Chemokines are grouped according to the number of cysteines and disulfide bonds in the molecule into C-X-C-, C-C, C, and CX 3 Cl Chemokine ligands (L) and receptors (R) and designated accordingly as CXCR1-7/CXCL1-16 and CCR1-10/CCL 1-28 (Allen et al., 2007) .”
Monocyte recruitment during infection and inflammation (2011, background): “In support of this model, monocyte recruitment is impaired by amino acid substitutions in CCL2 that prevent dimerization or association with glycosaminoglycan”
Orchestrating the orchestrators: chemokines in control of T cell traffic (2008, background): “213" language="eng" publish="issue" relation="no"> It is now firmly established that chemokines serve a central function in the immune system by coordinating the localization of immune cells in the body to generate an immune response at specific anatomic site”