Developmental origins of health and disease
By J. Shenai. Journal of Perinatology, 2007
632 citations recorded by the source corpus.
Subfields: General Health Professions, Pediatrics, Perinatology and Child Health
View Paper: https://www.semanticscholar.org/paper/5ce1a480128dda1a19c526d5b3a6012bd2709174
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In General Health Professions
In Pediatrics, Perinatology and Child Health
Citations in the corpus
- Perfluorinated Chemicals and Fetal Growth: A Study within the Danish National Birth Cohort (2007, background): “LBW has been associated with higher mortality and morbidity that is not restricted to early years of life (Glucksman and Hanson 2006), although this may reflect underlying causes of fetal growth impairment rather than LBW itself.”
- Maternal intake of vitamin D during pregnancy and risk of recurrent wheeze in children at 3 y of age. (2007, background): “The role of maternal diet on risk of asthma in offspring is a particularly intriguing topic given the growing evidence on the developmental origins of health and disease (13, 14) and the early age of asthma onset (15, 16).”
- Population Policies, Fertility, Women's Human Capital, and Child Quality (2007, neutral): “…to improved adult health status, reduced chronic health problems after a individual reaches age 50, and increased years of disability-free life in old age, controlling in some cases for later socioeconomic status ( Kannisto, et al. 1994; Finch and Crimmins, 2004; Gluckman and Hanson, 2006).”
- High Prevalence of Type 2 Diabetes and Pre-Diabetes in Adult Offspring of Women With Gestational Diabetes Mellitus or Type 1 Diabetes (2008, background): “Studies of developmental origins of health and disease have put focus on the possible role of intrauterine hyperglycemia in the pathogenesis of type 2 diabetes (1).”
- Effects of Late Gestational High Fat Diet on Body Weight, Metabolic Regulation and Adipokine Expression in Offspring (2013, neutral): “There is compelling evidence of a sizeable contribution of early-life influences on the risk of becoming an obese adolescent or adult, and of developing obesity-associated diseases in adulthood (6).”
- Epigenetics and the embodiment of race: Developmental origins of US racial disparities in cardiovascular health (2009, background): “This article does not comprehensively review the DOHaD or life-course epidemiology literatures, which have been the subject of extensive recent reviews (BenShlomo and Kuh, 2002; Gluckman and Hanson, 2006; Kuh et al., 2003; Smith, 2003).”
- The nature of nutrition: a unifying framework (2011, background): “For example, dietary-induced obesity in mothers and even grandmothers has a direct influence over the metabolism and risk of obesity in offspring (Barker 1998; McMillen and Robinson 2005; Gluckman and Hanson 2006a).”
- Etiologic Subtypes of Attention-Deficit/Hyperactivity Disorder: Brain Imaging, Molecular Genetic and Environmental Factors and the Dopamine Hypothesis (2007, methodology): “The DOHaD theory has been applied to explain the relationship of growth restriction to obesity and diabetes, as well as to cardiovascular disease and other physical disorders (see Gillman, 2005).”
- The Economics of Human Development and Social Mobility (2014, neutral): “…and life outcomes of children from disadvan- 11For example, Barker (1990) and Hales and Barker (1992) propose a “thrifty phenotype” hypothesis, now widely accepted, that reduced fetal growth is associated with a number of chronic conditions later in life (Gluckman and Hanson, 2005, 2006).”