Role of inflammatory mediators in the pathophysiology of acute respiratory distress syndrome

By M. Bhatia, S. Moochhala. Journal of Pathology, 2004

1,137 citations recorded by the source corpus.

Subfields: Critical Care and Intensive Care Medicine, Epidemiology, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/5a3b81e83bb52fa3d6c5ae3f985f5d15f6f6d8fc

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

1. M. Keel
2. O. Trentz

In Pulmonary and Respiratory Medicine

1. Brigham C. Willis
2. Z. Borok

Citations in the corpus

• Cytokines in Sepsis: Potent Immunoregulators and Potential Therapeutic Targets—An Updated View (2013, background): “TNF-α into experimental animals causes a syndrome that is largely indistinguishable from septic shock [107] and infusion of recombinant TNF-α into humans results in SIRS [108– 110].”
• n-3 polyunsaturated fatty acids, inflammation, and inflammatory diseases. (2006, neutral): “Acute respiratory distress syndrome (94)”
• Pathophysiology of polytrauma. (2005, background): “Examples of early organ dysfunction are the primary cerebral oedema after head injury, or the primary ARDS after thoracic injury.(15,18,95,120,137) The clinical manifestation of secondary MODS varies in affected organs and dysfunction severities.”
• Induction of epithelial-mesenchymal transition in alveolar epithelial cells by transforming growth factor-beta1: potential role in idiopathic pulmonary fibrosis. (2005, background): “:3) in lung injury 31,32 and its potential role in EMT led us to evaluate its ability to potentiate the effects of TGF-(cid:2) 1.”