Pathophysiological mechanisms of atrial fibrillation: a translational appraisal.

By Ulrich Schotten, S. Verheule, Paulus Kirchhof, A. Goette. Physiological Reviews, 2011

1,239 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine

View Paper: https://www.semanticscholar.org/paper/58aa09fb6abeefe70210dd4f48c3496bdf17141f

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In Cardiology and Cardiovascular Medicine

Citations in the corpus

2017 HRS/EHRA/ECAS/APHRS/SOLAECE expert consensus statement on catheter and surgical ablation of atrial fibrillation (2017, background): “For many years, three concepts competed to explain the mechanism of AF: multiple reentrant wavelets (Figure 3A), rapidly discharging automatic foci (Figure 3B), and a single reentrant circuit with fibrillatory conduction (Figure 3C).(194,195,196) Considerable progress has been made in defining the m”
Guidelines for the management of atrial fibrillation: the Task Force for the Management of Atrial Fibrillation of the European Society of Cardiology (ESC). (2010, background): “Pathophysiological changes as a consequence of atrial fibrillation After the onset of AF, changes of atrial electrophysiological properties, mechanical function, and atrial ultrastructure occur with different time courses and with different pathophysiological consequences.(18) Shortening of the atri”
2016 ESC Guidelines for the management of atrial fibrillation developed in collaboration with EACTS. (2016, neutral): “Multiple pathophysiological mechanisms can contribute to AF in obstructive sleep apnoea, including autonomic dysfunction, hypoxia, hypercapnia, and inflammation.(96,304) – 307 Obstructive sleep apnoea exaggerates intrathoracic pressure changes, which in itself and via vagal activation can provoke sh”
On Sampling Spatially-Correlated Random Fields for Complex Geometries (2019, background): “An example of the severity of these eﬀects is seen in the atria, whose tissue is a characterized by a complex structure of several ﬁbre bundles [15].”
The clinical profile and pathophysiology of atrial fibrillation: relationships among clinical features, epidemiology, and mechanisms. (2014, background): “AF-induced remodeling and contribute to the “AF begets AF ” phenomenon.(165,166)”
EHRA/HRS/APHRS/SOLAECE expert consensus on atrial cardiomyopathies: definition, characterization, and clinical implication. (2016, neutral): “endothelin-1 are linked to inflammatory and proarrhythmogenic atrial remodelling.(2,264) –266 This evidence suggests that inflammatory cell infiltration has an important role in promoting the creation of a substrate for AF, as a result of conduction heterogeneity and slowing, both in the setting of ”
A technique for measuring anisotropy in atrial conduction to estimate conduction velocity and atrial fibre direction (2018, neutral): “Changes that occur during AF that modify atrial conduction include down-regulation and lateralisation of connexins, deposition of collagen and interstitial fibrosis, as well as changes in atrial fibre direction, including fibre disarray [1,2].”