The clinical profile and pathophysiology of atrial fibrillation: relationships among clinical features, epidemiology, and mechanisms.
By Jason G. Andrade, P. Khairy, Dobromir Petkov Dobrev, Stanley Nattel. Circulation Research, 2014
1,077 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine
View Paper: https://www.semanticscholar.org/paper/32c1699a52211b1deb946b2a900e7fe3e8834e71
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cardiology and Cardiovascular Medicine
- G. Raskob
- Aaron M. Wendelboe
- Dobromir Petkov Dobrev
- Gerhard Hindricks
- Rahul Wadke
- A. Goette
- Gregory Y. H. Lip
- Stanley Nattel
Citations in the corpus
- Global Burden of Thrombosis: Epidemiologic Aspects. (2016, methodology): “For further reading on atrial fibrillation, please refer to the recent compendium edited by Andrade et al.(8) Because the name implies, VTE entails the formation of blood clots in the veins, most commonly in the deep veins of the calf and thigh, though ≤10% of clots may form in the upper extremities”
- 2017 HRS/EHRA/ECAS/APHRS/SOLAECE expert consensus statement on catheter and surgical ablation of atrial fibrillation (2017, background): “Two studies evaluated the use of early reablation on long-term freedom from AF in patients with ERAF.(141,142,143,989,993) In 302 consecutive patients with RF ablation for medically refractive AF, 151 experienced an ERAF, 61 of whom underwent reablation within the first month (e.”
- Atrial fibrillation. (2022, background): “5%).(13) However, as hypertension is very common, it has been estimated that 14% of all cases of atrial fibrillation are caused by hypertension.”
- EHRA/HRS/APHRS/SOLAECE expert consensus on atrial cardiomyopathies: definition, characterization, and clinical implication. (2016, positive): “Recently, atrial adipose tissue has emerged as a potential player in the pathophysiology of AF.(3,254) In addition to its paracrine effects,(192)”
- Role of the Autonomic Nervous System in Atrial Fibrillation: Pathophysiology and Therapy (2014, background): “Finally, structural remodeling is known to be an important contributor to AF persistence.(66) Increased Ca/calmodulin binding caused by β-adrenergic stimulation activates the protein phosphatase calcineurin (Figure 4).”
- Cellular and Molecular Electrophysiology of Atrial Fibrillation Initiation, Maintenance, and Progression (2014, background): “mechanisms is provided in another article in this compendium, along with an overview of AF pathophysiology.(15)”