Maintaining genome stability at the replication fork
By Dana Branzei, M. Foiani. Nature reviews. Molecular cell biology, 2010
820 citations recorded by the source corpus.
Subfields: Cell Biology, Molecular Biology, Oncology
View Paper: https://www.semanticscholar.org/paper/56e761078363c7a15ba8b3dab49a9ed24b7f1e89
How researchers see this paper
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In Cell Biology
- Stephen J. Elledge
- Debashish Chowdhury
- Alberto Ciccia
- T. Halazonetis
- D. Pellman
- Stephen P. Jackson
- Y. Fouad
- C. Aanei
In Molecular Biology
- Ryuji Hamamoto
- Yusuke Nakamura
- G. Toyokawa
- Hyun-soo Cho
- S. Hayami
- N. Dohmae
- Takehiro Suzuki
- David E. Neal
In Oncology
- Stephen J. Elledge
- Morgane Macheret
- Alberto Ciccia
- Debashish Chowdhury
- Bruce A. J. Ponder
- T. Halazonetis
- Ryuji Hamamoto
- Stephen P. Jackson
Citations in the corpus
- DNA replication stress as a hallmark of cancer. (2015, background): “DNA replication stress is a term that broadly defines impediments in DNA replication and generally includes stalling and collapse of DNA replication forks (68, 69).”
- Minichromosome Maintenance Protein 7 is a potential therapeutic target in human cancer and a novel prognostic marker of non-small cell lung cancer (2011, background): “A large number of molecular players, including minichromosome maintenance (MCM) proteins, are involved in DNA replication [6-8].”
- DNA breaks and chromosome pulverization from errors in mitosis (2012, background): “As expected, after S-phase entry, low-level DNA damage was detected in both the micronuclei and the primary nucleu”
- Nucleotide-resolution DNA double-strand breaks mapping by next-generation sequencing (2013, neutral): “captured on streptavidin beads (gray ovals) (3).”
- Chromatin replication and epigenome maintenance (2012, background): “Deregulation of DNA replication, including uncontrolled initiation and fork collapse, can promote DNA damage and genome instabilit”
- The DNA Damage Response: Making it safe to play with knives (2010, background): “Replication restart would leave behind the replication fork ssDNA gaps, which in yeast have been shown to be subsequently repaired by either translesion synthesis (TLS) or error-free postreplication repair (PRR) in a manner dependent on the RAD6/RAD18 ubiquitin ligase complex (Branzei and Foiani, 20”
- Stochastic mechano-chemical kinetics of molecular motors: A multidisciplinary enterprise from a physicist’s perspective (2012, background): “Moreover, we review some of the common causes for a temporary pause or permanent stall of a replication fork by either damage of the track or by the present of “blockage” on its way [1602].”
- Targeting hypoxia in cancer therapy (2011, background): “— on homologous recombination (HR) repair, which is required for the resolution of damage at the replication for”