Oligomerization of G-protein-coupled transmitter receptors
By Michel Bouvier. Nature Reviews Neuroscience, 2001
679 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Molecular Biology, Radiology, Nuclear Medicine and Imaging
View Paper: https://www.semanticscholar.org/paper/5078cf0a28076782be67b4c4a35167df58e26506
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cellular and Molecular Neuroscience
- Charles R. Yang
- Jeremy K. Seamans
- B. Bettler
- Torben Ott
- Andreas Nieder
- Michael J Frank
- Randall C. O’Reilly
- David E. Gloriam
In Molecular Biology
- Jeremy K. Seamans
- Charles R. Yang
- B. Bettler
- Torben Ott
- Andreas Nieder
- David E. Gloriam
- Oliver D. Howes
- Shitij Kapur
In Radiology, Nuclear Medicine and Imaging
Citations in the corpus
- The principal features and mechanisms of dopamine modulation in the prefrontal cortex. (2004, background): “This novel direct receptor-receptor oligomerization mechanism (Bouvier, 2001; Gazi et al., 2002) has also been shown for the D5/ GABAA receptor interaction (Liu et al.”
- International Union of Pharmacology. XXXIII. Mammalian γ-Aminobutyric AcidB Receptors: Structure and Function (2002, background): “In some cases they may be present as a single protein, whereas in others they may form homodimers (Bouvier, 2001).”
- The activation mechanism of class-C G-protein coupled receptors (2004, result): “As such, the elucidation of the activation mechanism of class-C GPCRs suggests that most, if not all GPCRs function as dimers, an idea supported by several recent studies (Bouvier, 2001; Baneres and Parello, 2003; Fotiadis et al., 2003; Liang et al., 2003).”
- Molecular Structure and Physiological Functions of GABAB Receptors (2004, background): “In addition to GABAB receptors, there are now several examples of GPCRs where heteromerization changes pharmacological and signaling properties (7, 43).”
- International Union of Pharmacology LVIII: Update on the P2Y G Protein-Coupled Nucleotide Receptors: From Molecular Mechanisms and Pathophysiology to Therapy (2006, background): “The missing numbers in the P2Y1–14 sequence represent GPCRs cloned from nonmammalian vertebrates or receptors for which a functional response to nucleotides has not yet been convincingly demonstrated.”