Whole-Genome Comparison of Mycobacterium tuberculosis Clinical and Laboratory Strains
By R. Fleischmann, D. Alland, Jonathan A. Eisen, L. Carpenter, Owen White, J. Peterson, R. Deboy, R. Dodson, M. Gwinn, Daniel H. Haft and 16 more. Journal of Bacteriology, 2002
737 citations recorded by the source corpus.
Subfields: Epidemiology, Infectious Diseases, Surgery
View Paper: https://www.semanticscholar.org/paper/4cd54941d1b78b7d84f45bc5ff92e44378a77442
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Citations in the corpus
- Versatile and open software for comparing large genomes (2004, background): “When two species are very similar, such as the two isolates of the Bacillus anthracis Ames strain sequenced at TIGR [31-33], then MUMmer is ideally suited for aligning the genomes. that comparison of anthrax isolates, only four single-nucleotide differences separated the two 5.3 Mbp main…”
- The Population Genetics of dN/dS (2008, background): “This observation holds for bacterial data [11,12,14,16,18], for viral samples isolated from a single host versus viral samples isolated from different hosts [13], for closely related viral samples versus distantly diverged samples [48], and for conspecific versus interspecific mammalian sequences [4”
- High Functional Diversity in Mycobacterium tuberculosis Driven by Genetic Drift and Human Demography (2008, neutral): “tuberculosis strain CDC1551 [66], M.”
- Prophages and bacterial genomics: what have we learned so far? (2003, background): “A few of the larger bacterial genome sequences, for example those of the high G+C Gram-positive bacteria such as Mycobacterium (4.4mbp) and Streptomyces (9.07mbp) have relatively few convincing prophages (Fleischmann et al., 2002; Bentley et al., 2002).”
- Ancient Origin and Gene Mosaicism of the Progenitor of Mycobacterium tuberculosis (2005, background): “the MTBC is consistent with recent expansion [4,10].”
- The Continuing Challenges of Leprosy (2006, methodology): “Such a decline has been documented, for example, for indicators of inflammation such as neopterin and iNOS (114, 233), as well as for cytokines and receptors more indicative of immunologic function such as soluble IL-2 receptor, IFN- , IL-6, IL-10, IL-12, and IL-13 (21, 233, 416).”
- Mycobacterium tuberculosis Pathogenesis and Molecular Determinants of Virulence (2003, background): “This is due to the development of many genetic methods, mainly by the Gicquel and Jacobs laboratories (109, 218), and to the DNA sequencing and annotation of the M. tuberculosis H37Rv genome (53) and those of related mycobacteria that have been or are currently being completed by The Institute for G”