Anti-Hu-associated paraneoplastic encephalomyelitis: analysis of 200 patients.
By F. Graus, F. Keime-Guibert, R. Reñé, B. Benyahia, T. Ribalta, Carlos Ascaso, Geòrgia Escaramís, Jean-Yves Delattre. Brain : a journal of neurology, 2001
896 citations recorded by the source corpus.
Subfields: Hematology, Neurology, Pulmonary and Respiratory Medicine
View Paper: https://www.semanticscholar.org/paper/4c62c1c911cea0c67d8d33cc84bc2580b1efa75f
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In Neurology
Citations in the corpus
- Chemotherapy‐induced neuropathy (2008, background): “Differential diagnosis and laboratory studies The major differential diagnosis is between CIN and paraneoplastic sensory ganglioneuropathy (Voltz et al., 1997; Graus et al., 2001).”
- Potassium channel antibody-associated encephalopathy: a potentially immunotherapy-responsive form of limbic encephalitis. (2004, neutral): “…reported cases are associated with speci®c autoantibodies; mainly to Hu in patients with lung cancer (Dalmau et al., 1992; Alamowitch et al., 1997; Graus et al., 2001), to Ma2 in patients with testicular tumours (Voltz et al., 1999) or to CRMP5/CV2 in patients with thymomas (Antoine et al.,…”
- Paraneoplastic anti–N‐methyl‐D‐aspartate receptor encephalitis associated with ovarian teratoma (2007, background): “Despite the severity of the symptoms, paraneoplastic anti-NMDAR encephalitis has a better prognosis than most other paraneoplastic encephalitides.(3,36) Nine of 12 patients significantly recovered and 7 returned to their jobs.”
- Clinical analysis of anti-Ma2-associated encephalitis. (2004, methodology): “Only four out of 200 (2%) patients with anti-Hu-associated encephalomyelitis had improvement of CNS symptoms, and none of 34 patients with anti-Yoassociated cerebellar degeneration had neurological improvement after oncological treatment, immunotherapy or both (Rojas et al., 2000; Graus et al., 2001”
- Immunopathology of autoantibody-associated encephalitides: clues for pathogenesis. (2012, background): “Approximately 60% of patients with these syndromes have serum and CSF antibodies to intracellular antigens, often even intranuclear antigens such as Hu, Ma, Yo or amphiphysin (Saiz et al., 1999; Graus et al., 2001; Dalmau and Rosenfeld, 2008).”