Potassium channel antibody-associated encephalopathy: a potentially immunotherapy-responsive form of limbic encephalitis.

By A. Vincent, Camilla J. Buckley, Jonathan M. Schott, Ian Baker, B. Dewar, N. Detert, L. Clover, A. Parkinson, C. Bien, Salah Omer and 3 more. Brain : a journal of neurology, 2004

1,042 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Molecular Biology, Neurology

View Paper: https://www.semanticscholar.org/paper/75afcb87c91b88f3cd9ead173035a4462170e343

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. Hans Lassmann
2. L. R. Squire
3. K. Jellinger
4. Y. Dudai
5. Omar Alcides Lizardo
6. F. G. Ashby
7. Eric
8. Yeh

In Molecular Biology

1. Hans Lassmann
2. Michael Barnett
3. K. Jellinger

In Neurology

1. Hans Lassmann
2. J. Dalmau
3. L. R. Squire
4. K. Jellinger

Citations in the corpus

• The neuroscience of remote memory. (2007, background): “Yet, when one examines these studies, one finds that investigators have usually described damage that extends beyond the medial temporal lobe: KC was a case of head trauma with significant damage in frontal, parietal and occipital cortices [22]; VC [16] has evidence of hypometabolism in the right th”
• Faciobrachial dystonic seizures precede Lgi1 antibody limbic encephalitis (2011, background): “%) experienced FBDS prior to the development of the amnesia and confusion that characterize LE.”
• Recommended diagnostic criteria for paraneoplastic neurological syndromes (2004, background): “11 The presence of high levels of voltage gated potassium channel (VGKC) antibodies may suggest the diagnosis of idiopathic limbic encephalitis 12 but these antibodies are also found in a few patients with a paraneoplastic origin, 13 so the test should not be used to exclude a paraneoplastic cause…”
• Immunopathology of autoantibody-associated encephalitides: clues for pathogenesis. (2012, background): “…disorders associated with surface antigens, such as encephalitis with antibodies to the VGKC-complex or NMDAR, often respond well to treatment (Vincent et al., 2004; Malter et al., 2010; Dalmau et al., 2011) and may be mediated by antibody-binding, internalization and loss of the target…”