ADAMTS-7 Inhibits Re-endothelialization of Injured Arteries and Promotes Vascular Remodeling Through Cleavage of Thrombospondin-1
By T. Kessler, Lu Zhang, Ziyi Liu, X. Yin, Yaqian Huang, Yingbao Wang, Yi Fu, M. Mayr, Qing Ge, Qingbo Xu and 8 more. 2015
138 citations recorded by the source corpus.
Subfields: Immunology and Allergy, Molecular Biology, Rheumatology
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In Immunology and Allergy
In Molecular Biology
Citations in the corpus
- ADAMTS proteins in human disorders (2018, background): “Instead, thrombospondin appears to be the relevant substrate for this effect, since the impact of Adamts7 deficiency on re-endothelialization was not seen in Tsp1−/− mice [85].”
- Upregulation of ADAMTS-7 and downregulation of COMP are associated with aortic aneurysm (2017, unknown): “It has previously been demonstrated that increased MMP‑9 results in VSMCs apoptosis and collagen degeneration in the most frequently occurring form of thoracic aortic aneurysm (18,19).”