Inflammatory mediators and the failing heart: past, present, and the foreseeable future.
By Douglas L. Mann. Circulation Research, 2002
1,028 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Pathology and Forensic Medicine, Pharmacology
View Paper: https://www.semanticscholar.org/paper/4a1e9efad0e3b8940fcd352a5a497b00f8518213
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cardiology and Cardiovascular Medicine
- Sumanth D. Prabhu
- Yibin Wang
- Nikolaos G. Frangogiannis
- S. von Haehling
- S. Anker
- T. Force
- K. Weeks
- Bianca C. Bernardo
In Pathology and Forensic Medicine
In Pharmacology
Citations in the corpus
- Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale. (2010, background): “However, given their importance in cardiac pathologies, the reader is directed to a number of excellent reviews regarding these topics (33, 131, 136, 160, 259, 325, 346).”
- Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets (2015, background): “However, evidence suggests that the initial short-term inflammatory response is an adaptive response, which is important for cardiac repair (Mann 2002).”
- The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis. (2016, methodology): “After healing, in both humans and animal models with MI, a subset of those afflicted will exhibit late progressive ventricular dilatation and HF, a state characterized by chronic inflammation (Figure 5).(80) Among other variables, both larger infarcts(253) and greater initial inflammatory activation”
- Inflammatory mediators in chronic heart failure: an overview (2004, background): “Chronic heart failure (CHF) represents a major public health burden, and its prognosis is comparable to that of different malignant diseases. Our understanding of CHF has developed from the rather simplistic model of mere pump failure to that of a multisystem disorder which affects not only the card”
- Mitochondrial DNA That Escapes from Autophagy Causes Inflammation and Heart Failure (2012, methodology): “To identify the source of IL-1b and IL-6, we performed in situ hybridization analysis in heart sections.”