The hypersensitive response; the centenary is upon us but how much do we know?
By L. Mur, P. Kenton, A. Lloyd, H. Ougham, E. Prats. Journal of Experimental Botany, 2008
693 citations recorded by the source corpus.
Subfields: Cell Biology, Epidemiology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/490af1266844e31461addb94bce633016536a18a
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Citations in the corpus
- Stress-induced electrolyte leakage: the role of K+-permeable channels and involvement in programmed cell death and metabolic adjustment. (2014, background): “PCD can help plants to survive under pathogen attack, herbicide treatment, and heavy metal stress (Cutler and Somerville, 2005; Mur et al., 2008).”
- Worldwide Research on Plant Defense against Biotic Stresses as Improvement for Sustainable Agriculture (2018, background): “This type of resistance is named effector-triggered immunity (ETI) and often activates hypersensitive responses (HR) that include programmed cell death in infected cells and the surrounding areas [18].”
- The Arabidopsis NPR1 protein is a receptor for the plant defense hormone salicylic acid. (2012, background): “Interestingly, HR possesses most of the typical features of both the non-programmed cell death (mitochondrial swelling of oncosis) and the PCD (cytoplasmic shrinkage, chromatin condensation of apoptosis, and vacuolization of autophagy) (Mur et al., 2008).”
- Programmed cell death in the plant immune system (2011, methodology): “transcriptional reprogramming and synthesis of antimicrobial compounds.(23) Compared with MTI, ETI is typically an”
- Reactive oxygen species and antioxidant machinery in abiotic stress tolerance in crop plants. (2010, background): “Recent researches have linked chloroplast-produced ROS with the hypersensitive response [60].”
- Salicylic Acid, a multifaceted hormone to combat disease. (2009, background): “One of the earliest responses observed after pathogen attack is the OB, which involves a rapid increase in ROS such as superoxide (O2−) and H2O2 in the apoplast (see sidebar, Accumulation of Reactive Oxygen Species During the Oxidative Burst) (37, 70, 107, 134, 148).”