Misdiagnosis of hereditary amyloidosis as AL (primary) amyloidosis.

By H. Lachmann, D. Booth, S. Booth, A. Bybee, J. Gilbertson, J. Gillmore, M. Pepys, P. Hawkins. New England Journal of Medicine, 2002

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Systemic Cardiac Amyloidoses: Disease Profiles and Clinical Courses of the 3 Main Types (2009, neutral): “Left bundle-branch block, n/N (%) 6/146 (4)* 4/60 (7)‡ 6 (40) 0.”
Noninvasive etiologic diagnosis of cardiac amyloidosis using 99mTc-3,3-diphosphono-1,2-propanodicarboxylic acid scintigraphy. (2005, background): “isdiagnosis rate between familial and AL amyloidosis (7).”
Definition of organ involvement and treatment response in immunoglobulin light chain amyloidosis (AL): A consensus opinion from the 10th International Symposium on Amyloid and Amyloidosis (2005, background): “The presence of a monoclonal gammopathy should not be used as the only evidence of AL, because a small fraction of patients with familial (AF), secondary (AA), and senile systemic amyloidosis (SSA) will have an incidental monoclonal gammopathy [35] and evidence of clonal plasma cell dyscrasia associ”
High-Dose Melphalan and Autologous Stem-Cell Transplantation in Patients with AL Amyloidosis: An 8-Year Study (2004, neutral): “No No 9/22 (41) 17/29 (59) No Yes 6/34 (18) 23/69 (33) Yes No 3/3 (100) 10/11 (91)”