Noninvasive etiologic diagnosis of cardiac amyloidosis using 99mTc-3,3-diphosphono-1,2-propanodicarboxylic acid scintigraphy.
By E. Perugini, P. Guidalotti, F. Salvi, Robin M. T. Cooke, C. Pettinato, L. Riva, O. Leone, M. Farsad, P. Ciliberti, L. Bacchi-Reggiani and 3 more. Journal of the American College of Cardiology, 2005
892 citations recorded by the source corpus.
Subfields: Molecular Biology, Pulmonary and Respiratory Medicine
View Paper: https://www.semanticscholar.org/paper/cabd9b7a9863914cfb2daefb6c7dfcd783aa17ef
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In Molecular Biology
Citations in the corpus
- Nonbiopsy Diagnosis of Cardiac Transthyretin Amyloidosis (2016, methodology): “Images were acquired with lowenergy, high-resolution collimators and a scan speed of 10 cm/ min (99mTc-DPD and 99mTc-HMDP) or for a total of 750 000 counts (99mTc-PYP).24 Cardiac retention of all 99mTc-DPD and 99mTc-HMDP scans was defined by a single reader at each center according to the grading de”
- Guideline of transthyretin-related hereditary amyloidosis for clinicians (2013, background): “There is increasing evidence that Tc-DPD scintigraphy can image amyloid deposition in the myocardium with a greater sensitivity among patients with TTR-related cardiac amyloidosis (both mutant and wild-type) than immunoglobulin AL-related amyloidosis, and that, compared with a small series of normal”