Increased dietary intake of ω-3-polyunsaturated fatty acids reduces pathological retinal angiogenesis

By K. Connor, J. Sangiovanni, Chatarina Lofqvist, C. Aderman, Jing Chen, A. Higuchi, Song Hong, Elke A. Pravda, Sharon F Majchrzak, D. Carper and 6 more. Nature Medicine, 2007

725 citations recorded by the source corpus.

Subfields: Biochemistry, Nutrition and Dietetics, Radiology, Nuclear Medicine and Imaging

View Paper: https://www.semanticscholar.org/paper/448ecdb1d2df0aaf333ed0a7821fc98324b05f46

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Biochemistry

1. C. Serhan
2. R. Ji
3. Rong Yang
4. Maiken Nedergaard
5. P. Calder
6. B. Levy
7. T. Kern
8. Johnny Tang

In Nutrition and Dietetics

1. C. Serhan
2. R. Ji
3. Rong Yang
4. B. Levy
5. P. Calder
6. R. Medzhitov
7. T. Kern
8. Johnny Tang

In Radiology, Nuclear Medicine and Imaging

1. Maiken Nedergaard

Citations in the corpus

• Glia and pain: Is chronic pain a gliopathy? (2013, background): “The receptors of these mediators, such as ChemR23 (RvE1) and ALX (RvD1 and LXA4) are widely expressed in neurons, glia, and immune cells [39,114,210,231].”
• Resolvins RvE1 and RvD1Attenuate Inflammatory Pain via Central and Peripheral Actions (2010, positive): “Recently, resolvins, a unique family of lipid mediators, including RvE1 and RvD1 derived from omega-3 polyunsaturated fatty acid, have shown marked potency in treating disease conditions associated with inflammatio”
• Quantification of oxygen-induced retinopathy in the mouse: a model of vessel loss, vessel regrowth and pathological angiogenesis (2009, background): “Phase I is also precipitated after premature birth by the loss of factors normally provided by the mother in utero , such as insulin-like growth factor (IGF-1) and ωω-polyunsaturated fatty acid”
• The mouse retina as an angiogenesis model. (2010, methodology): “Work from the Chan laboratory found that -3 fatty acids can slow and even reverse the progression of AMD-like neovascular lesions in these models,(180) thus confirming earlier results obtained in epidemiologic AMD studies(181) and in the OIR mouse model.(127)”