Environmental signaling: what embryos and evolution teach us about endocrine disrupting chemicals.
By J. Mclachlan. Endocrine reviews, 2001
635 citations recorded by the source corpus.
Subfields: Genetics, Health, Toxicology and Mutagenesis, Pollution
View Paper: https://www.semanticscholar.org/paper/436fa2b3e2676d2265acd54c400c1ee97b4e1eda
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Citations in the corpus
- Evolution of dioecy and sex chromosomes via methylation driving Muller's ratchet (2003, background): “Recent work indicates that sex hormones determine sex via heritable changes in methylation patterns (McLachlan, 2001).”
- The timing of normal puberty and the age limits of sexual precocity: variations around the world, secular trends, and changes after migration. (2003, background): “Also, the possible relationship between sexual precocity and fetal or perinatal exposure to EDCs is raising the issue of the period of exposure during development, a possibly critical parameter (263, 264).”
- The Estrogenic Effect of Bisphenol A Disrupts Pancreatic β-Cell Function In Vivo and Induces Insulin Resistance (2005, background): “106 VOLUME 114 | NUMBER 1 | January 2006 • Environmental Health Perspectives”
- Overview of Nomenclature of Nuclear Receptors (2006, background): “In this respect, many plant and industrial chemicals, including pesticides, plastic components, and xenobiotic drugs, have been found to bind to and thereby mimic, block, or otherwise disrupt the natural activity of NRs (McLachlan, 2001).”