Normal high density lipoprotein inhibits three steps in the formation of mildly oxidized low density lipoprotein: step 1.
By M. Navab, S. Hama, C. Cooke, G. Anantharamaiah, M. Chaddha, L. Jin, G. Subbanagounder, K. F. Faull, S. T. Reddy, N. Miller and 1 more. Journal of Lipid Research, 2000
596 citations recorded by the source corpus.
View Paper: https://www.semanticscholar.org/paper/411da8356d2fee631663c912d7e472369f2b51db
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Citations in the corpus
- Apolipoprotein A-I is a selective target for myeloperoxidase-catalyzed oxidation and functional impairment in subjects with cardiovascular disease. (2004, background): “Several functional activities associated with HDL and apoA-I are believed to promote these beneficial effects, including both facilitation of reverse cholesterol transport and additional cardioprotective antioxidant and antiinflammatory functions (49, 68, 69).”
- Effects of exercise and diet on chronic disease. (2005, background): “These investigators observed that the HDL from the patients was not protective against LDL oxidation (270).”
- HDL and cardiovascular disease: atherogenic and atheroprotective mechanisms (2011, background): “Apo A-I by itself reduces the proinflammatory effect of oxidized lipids”
- Functionally Defective High-Density Lipoprotein: A New Therapeutic Target at the Crossroads of Dyslipidemia, Inflammation, and Atherosclerosis (2006, background): “…of HDL can be ascribed to apoA-I which can prevent and/or delay LDL oxidation by removing oxidized PL, including 1- palmitoyl-2(5-oxovaleroyl)-sn-glycero-3-phosphorylcholine and 1-palmitoyl-2-glutaroyl-sn-glycero-3phosphorylcholine, from LDL and from arterial wall cells (Navab et al., 2000a,b).”
- Antiinflammatory Properties of HDL (2004, methodology): “This limits the oxidation of phospholipids within LDL, along with the subsequent inflammatory response of atherosclerosis [35,36].”
- Ambient Particulate Pollutants in the Ultrafine Range Promote Early Atherosclerosis and Systemic Oxidative Stress (2008, methodology): “Although the methodology used is sensitive and specific for the determination of MDA,28 there are several limitations in this assay in reflecting the degree of lipid peroxidation, as reviewed by Janero et al,29, such as: (1) MDA yield as a result of lipid peroxidation varies with the nature of the p”