Genetic regulation of tomato fruit ripening and development and implementation of associated genomics tools

By James J. Giovannoni, S. El-Rakshy. 2005

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Cell wall metabolism in fruit softening and quality and its manipulation in transgenic plants (2001, background): “Fruit of rin (ripening inhibitor) and nor (non-ripening) lack the normal climacteric and produce only basal levels of ethylene, do not develop carotenes and lycopene, and soften very slowly (Tigchelaar et al., 1978).”
Climacteric fruit ripening: Ethylene-dependent and independent regulation of ripening pathways in melon fruit (2008, background): “In the tomato, the rin mutation [19] that encodes a MADS box type transcription factor and regulates the ripening process confers ethylene insensitivity to the fruit [20] and has been used for generating long shelf-life commercial varieties.”
Comprehending crystalline β-carotene accumulation by comparing engineered cell models and the natural carotenoid-rich system of citrus (2011, unknown): “To confirm these results further, the expression profiles of regulatory genes previously described as involved in the ripening of tomato fruit, such as RIPENING INHIBITOR (RIN), COLORLESS NON RIPENING (CNR), NON-RIPENING (NOR), and NEVER RIPE (NR) were analysed (Tigchelaar et al., 1978; Wilkinson et”
Molecular biology of ethylene during tomato fruit development and maturation (2008, background): “The fruit produced by these mutants fail to display normal ripening phenotypes including ethylene synthesis, increased respiration, carotenoid accumulation, softening, and aroma volatile production [110,111].”