Dynamic interactions of cortactin and membrane type 1 matrix metalloproteinase at invadopodia: defining the stages of invadopodia formation and function.
By V. Artym, Ying Zhang, F. Seillier-Moiseiwitsch, Kenneth M. Yamada, S. Mueller. Cancer Research, 2006
616 citations recorded by the source corpus.
Subfields: Cancer Research, Cell Biology, Immunology and Allergy
View Paper: https://www.semanticscholar.org/paper/3e56e58d10e0ab851384bbafa6a291a7b1513743
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cancer Research
- Peter Friedl
- Christopher M. Overall
- Kenneth M. Yamada
- Gerhard Christofori
- Katarina Wolf
- Mahmut Yilmaz
- Erik Sahai
- Stephanie Alexander
In Cell Biology
- Kenneth M. Yamada
- Peter Friedl
- Katarina Wolf
- Gerhard Christofori
- Mahmut Yilmaz
- A. Berrier
- Stephanie Alexander
- R. Weinberg
In Immunology and Allergy
- Peter Friedl
- Christopher M. Overall
- Kenneth M. Yamada
- Gerhard Christofori
- A. Berrier
- Mahmut Yilmaz
- Stephanie Alexander
- Erik Sahai
Citations in the corpus
- The 'ins' and 'outs' of podosomes and invadopodia: characteristics, formation and function (2011, neutral): “The maturation process for podosomes and invadopodia involves the recruitment and activation of multiple pericellular proteases that facilitate ECM degradatio”
- EMT, the cytoskeleton, and cancer cell invasion (2009, background): “The formation of invadopodia follows distinct steps [194].”
- Multi-step pericellular proteolysis controls the transition from individual to collective cancer cell invasion (2007, background): “Consequently, adherent cells degrade ECM proteins at focal adhesion and motility structures including lamellae, pseudopodia and invadopodi”
- Cell–matrix adhesion (2007, background): “Because of their matrix remodeling properties, invadopodia are suggested to promote tumor cell invasion (Artym et al., 2006; Weaver, 2006; Linder, 2007).”