Sensitization in patients with painful knee osteoarthritis

By L. Arendt-Nielsen, H. Nie, M. Laursen, B. Laursen, P. Madeleine, O. Simonsen, T. Graven‐Nielsen. Pain, 2010

1,006 citations recorded by the source corpus.

Subfields: Pharmacology, Physiology, Rheumatology

View Paper: https://www.semanticscholar.org/paper/3d24702a1f1cfb340c886d6d49167701a35f90be

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Pharmacology

1. Clifford J. Woolf
2. R. Ji
3. Arne May

In Physiology

1. Francis J. Keefe
2. L. Porter
3. H. Schubiner
4. A. Caño
5. M. Lumley
6. J. L. Cohen
7. A. M. Radcliffe
8. Clifford J. Woolf

Citations in the corpus

• Central sensitization: Implications for the diagnosis and treatment of pain (2010, background): “While the degree of sensitization correlated with the pain, it did not correlate with radiological findings, leading to the conclusion that central sensitization is an important contributor to knee OA pain [7].”
• Pain and Emotion: A Biopsychosocial Review of Recent Research (2011, background): “For example, central sensitization develops after peripheral damage such as osteoarthritis (Arendt-Nielsen et al., 2010) or neck injuries (Banic et al., 2004).”
• Descending pain modulation and chronification of pain (2014, neutral): “Dysfunction of endogenous pain inhibition has been demonstrated in patients with fibromyalgia, irritable bowel syndrome, temporomandibular disorder, osteoarthritis, chronic pancreatitis and rheumatoid arthritis, and has reportedly been used to predict postsurgical pain [112 ■ -116 ■ ,117,118].”