Modulation of A-type potassium channels by a family of calcium sensors
By W. F. An, M. Bowlby, M. Betty, Jie Cao, H. Ling, Grace Mendoza, Joe Hinson, Karen Mattsson, B. Strassle, J. Trimmer and 1 more. Nature, 2000
1,010 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Cellular and Molecular Neuroscience, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/3c154a2dcc94956788e5455cde0b50e45645b756
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cardiology and Cardiovascular Medicine
In Cellular and Molecular Neuroscience
- William A. Catterall
- R. Kass
- J. Nerbonne
- F. H. Yu
- T. Scheuer
- Vladimir Yarov-Yarovoy
- K. Konoki
- Sandrine Cestèle
In Molecular Biology
- William A. Catterall
- J. Nerbonne
- R. Kass
- F. H. Yu
- T. Scheuer
- Vladimir Yarov-Yarovoy
- K. Konoki
- Sandrine Cestèle
Citations in the corpus
- Molecular physiology of cardiac repolarization. (2005, background): “x-encoded Kv channels, slows current inactivation, speeds recovery from inactivation and shifts the voltage dependence of channel activation (21, 193, 195).”
- Voltage-gated ion channels and gating modifier toxins. (2007, background): “…the common features of their structure and function, presents the sodium channel as a primary example of neurotoxin action on ion channels, and describes the voltage-sensor trapping model for gating modifier toxin action, which may be a common mechanism of polypeptide toxin action on ion channels.”