Peritoneal dialysis and epithelial-to-mesenchymal transition of mesothelial cells.

By María Yáñez-Mó, E. Lara-Pezzi, R. Selgas, M. Ramírez-Huesca, C. Domı́nguez-Jiménez, J. Jiménez-Heffernan, A. Aguilera, J. Sánchez-Toméro, M. Bajo, V. Alvarez and 6 more. New England Journal of Medicine, 2003

775 citations recorded by the source corpus.

Subfields: Infectious Diseases, Nephrology, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/3bb2ed51bc3d1ed9713b3a7d43901fdcdfbc82cf

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Citations in the corpus

Epithelial-mesenchymal transitions in development and disease. (2009, background): “also converted to mesenchyme in patients that receive ambulatory peritoneal dialysis and that develop peritoneal fibrosis (Yanez-Mo et al., 2003), a process that involves the MAPK pathway and Snail1 activation (Strippoli et al.”
Visualizing stromal cell dynamics in different tumor microenvironments by spinning disk confocal microscopy (2008, methodology): “Inhibition of ERK or NF-κB restores the epithelial phenotype in transdifferentiated MCs from peritoneal effluent EMT by peritoneal MCs in PD patients is linked to PM dysfunction (Aroeira et al., 2007; Yañez Mo et al., 2003; Aroeira et al., 2005; Del Peso et al., 2008).”
Snail blocks the cell cycle and confers resistance to cell death. (2004, background): “…the present study), in the mitotic arrest in the cells of the ventral furrow during Drosophila gastrulation (Foe 1989), the low proliferation in the invasive front of carcinomas (Jung et al. 2001), and the link between high p21 expression and poor prognosis in breast carcinomas (Yang et al. 2003).”
The Snail genes as inducers of cell movement and survival: implications in development and cancer (2005, background): “Thus, it not surprising to find that SNAIL1 is activated in the mesothelial cells of patients during the secondary fibrosis that is associated with prolonged peritoneal dialysis (Yáñez-Mo et al., 2003).”