Immunobiology of the TAM receptors
By G. Lemke, C. Rothlin. Nature reviews. Immunology, 2008
852 citations recorded by the source corpus.
Subfields: Immunology, Pharmacology
View Paper: https://www.semanticscholar.org/paper/3b7e49bca86842f4ebb9a5c6a4cc6d574ada9016
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In Immunology
Citations in the corpus
- Apoptotic cells promote their own clearance and immune tolerance through activation of LXR (2009, neutral): “Interestingly, TAM receptors have recently been linked to the suppression of TLR signaling, revealing one mechanism by which apoptotic cell clearance and immune tolerance are coupled (Lemke and Rothlin, 2008; Rothlin et al., 2007).”
- The role of defective clearance of apoptotic cells in systemic autoimmunity (2010, background): “The absence of these receptors is therefore thought to cause both the accumulation of uncleared apoptotic cells and the production of adjuvant cytokines, both of which occur in the severe autoimmune phenotype of TAM-defective animals”
- Focus on Extracellular Vesicles: Introducing the Next Small Big Thing (2016, background): “, TIM receptor family (T-cell-transmembrane/immunoglobulin/mucin) and stabilin-2 [89,90], or via bridging molecules such as soluble thrombospondins, milk fat globule epidermal growth factor 8 (MFG-E8), which links to integrins αvβ3 and αvβ5, or growth arrest-specific gene 6 (Gas6) and protein S (a v”
- Pathophysiology of Myocardial Infarction (2015, neutral): “(219), receptor interacting protein 3 (RIP3), a kinase that inhibits RIP1-induced NF-κB signaling (252), activating transcription factor 3, the autophagy-related molecule Atg16L1,”