Epstein–Barr virus: 40 years on
By L. Young, A. Rickinson. Nature Reviews. Cancer, 2004
2,275 citations recorded by the source corpus.
Subfields: Infectious Diseases, Oncology, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/3a36895c14af9b0da35cb703687aafa143915e12
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In Oncology
Citations in the corpus
- Constitutive AP-1 Activity and EBV Infection Induce PD-L1 in Hodgkin Lymphomas and Post-transplant Lymphoproliferative Disorders: Implications for Targeted Therapy (2012, background): “nism for the observed expression of PD-L1 in other malignancies with constitutive AP-1 activity such as anaplastic large cell lymphoma (7, 46, 47) and melanoma (48) and bases for further analyses of the PD-1 pathway in additional EBV-associated tumors (2, 49).”
- HUMAN VIRAL ONCOGENESIS: A CANCER HALLMARKS ANALYSIS (2014, background): “In immunocompetent individuals Lat III B-cells are eliminated by the immune system (Thorley-Lawson, 2001; Young and Rickinson, 2004) (Figure 2).”
- EBV Positive Mucocutaneous Ulcer – A study of 26 cases associated with various sources of immunosuppression (2010, background): “Key molecular pathways controlling the cell cycle, such as NF-kB, are activated and virus-induced cytokines exert paracrine proliferative effects.(28,35,38,70) In physiologic circumstances the inherent propensity of EBV to induce B-cell proliferation is counterbalanced by complex immunologic interac”