Oral corticosteroids and fracture risk: relationship to daily and cumulative doses.
By T. V. Staa, H. Leufkens, L. Abenhaim, B. Zhang, Chris Cooper. Rheumatology, 2000
719 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Orthopedics and Sports Medicine
View Paper: https://www.semanticscholar.org/paper/368dcfb1d836475f1d6bfdfb65b8a51e407bff82
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In Endocrinology, Diabetes and Metabolism
In Orthopedics and Sports Medicine
- Chris Cooper
- Eugene V. McCloskey
- J. Kanis
- Nicholas C. Harvey
- H. Johansson
- J. Reginster
- R. Rizzoli
- on behalf of the International Osteoporosis Foundation
Citations in the corpus
- UK clinical guideline for the prevention and treatment of osteoporosis (2017, background): “use of glucocorticoids is, however, not solely dependent upon bone loss and BMD-independent risks have been identified [36, 37] (Evidence level 1a).”
- The clinician’s guide to prevention and treatment of osteoporosis (2022, background): “Glucocorticoids cause a dose-dependent loss of BMD in the spine and hip, with the greatest loss in vertebral trabecular bone [274].”
- Pharmacokinetics and Pharmacodynamics of Systemically Administered Glucocorticoids (2005, background): “They inhibit dose compared with the cumulative dose.[221] Skin the differentiation and antigen presentation of atrophy is due to suppression of cutaneous cell prolimacrophages and dendritic cells[184,207] and thus supferation and protein synthesis (e.”