Artificial sweeteners induce glucose intolerance by altering the gut microbiota
By Jotham Suez, T. Korem, D. Zeevi, Gili Zilberman-Schapira, Christoph A. Thaiss, O. Maza, D. Israeli, N. Zmora, S. Gilad, A. Weinberger and 7 more. Nature, 2014
1,923 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Molecular Biology, Nutrition and Dietetics
View Paper: https://www.semanticscholar.org/paper/3445145760bd14e7d3b38d590568ec4f167d834c
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In Molecular Biology
Citations in the corpus
- Personalized Nutrition by Prediction of Glycemic Responses. (2015, methodology): “Illumina compatible libraries were prepared as described (Suez et al., 2014).”
- The gut microbiota: a major player in the toxicity of environmental pollutants? (2016, background): “The microbiota of these four responders changed markedly during the intervention period and clustered differently from the one of the three non-responders, in whom blood glucose levels were not affected post-saccharin treatment.(103) These results suggest that human individuals feature a personalise”
- Antibiotic-Induced Changes in the Intestinal Microbiota and Disease. (2016, background): “Consumption of artificial sweeteners in mice and humans has been shown to lead to deregulation of the intestinal microbiota, with altered glycan degradation capacity promoting decreased glucose tolerance, a pre-diabetic condition [58].”
- Personalized Gut Mucosal Colonization Resistance to Empiric Probiotics Is Associated with Unique Host and Microbiome Features. (2018, neutral): “, 2015) and synthetic food supplements (Suez et al., 2014).”
- Gut Microbiome: Profound Implications for Diet and Disease (2019, background): “In humans, the average bacterial-epithelial distance of closest bacteria correlates inversely with BMI, fasting glucose levels, and hemoglobin A1c [271].”
- The Western Diet–Microbiome-Host Interaction and Its Role in Metabolic Disease (2018, neutral): “Effects from rodent studies include altered composition of microbes leading to impaired glucose tolerance [28,59,60], as well as increased pro-inflammatory potential [61,62] and liver inflammation [61].”