The Y-family DNA polymerase κ (pol κ) functions in mammalian nucleotide-excision repair

By T. Ogi, A. Lehmann. Nature Cell Biology, 2006

186 citations recorded by the source corpus.

Subfields: Molecular Biology, Pathology and Forensic Medicine

View Paper: https://www.semanticscholar.org/paper/32add1f75c572d4805a987294bf25536d003d95e

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Citations in the corpus

• Mechanisms of DNA damage, repair and mutagenesis (2017, unknown): “For example, REV1 is very important in mouse cells for synthe- sis across post-replicative gaps where REV1 gets recruited to the gaps by the 50-end, unlike the gap-filling step of NER, where POL j is the polymerase of choice [Ogi and Lehmann, 2006; de Groote et al., 2011; Sale, 2013].”
• Nucleotide excision repair in eukaryotes. (2013, background): “The first unexpected finding was the involvement of the error-prone translesion synthesis polymerase Pol k in the repair synthesis step (Ogi and Lehmann 2006).”