Conduction Slowing and Sudden Arrhythmic Death in Mice With Cardiac-Restricted Inactivation of Connexin43

By D. Gutstein, G. Morley, H. Tamaddon, D. Vaidya, M. Schneider, Ju Chen, K. Chien, H. Stuhlmann, G. Fishman. Circulation Research, 2001

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Engineering Adolescence: Maturation of Human Pluripotent Stem Cell-derived Cardiomyocytes (2014, background): “It is worth mentioning these values are close to the conduction velocity of neonatal mouse hearts (≈29 cm/s),(106) whereas adult mouse hearts has a conduction of 62 cm/s.(107) The cardiomyocytes produced significant contractile forces ≤2 mN.”
Gap junctions and the connexin protein family. (2004, background): “Some of these studies [24,25] will be discussed later in this Spotlight Issue.”
Structural and Functional Diversity of Connexin Genes in the Mouse and Human Genome (2002, methodology): “Gutstein et al. (2001) have also generated floxed Cx43 mice and crossed them with α-myosin heavy chain promotor Cre mice to achieve cardiomyocyte-specific deletion of Cx43.”