The distinct morphogenic states of Candida albicans.
By P. Sudbery, N. Gow, J. Berman. Trends in Microbiology, 2004
928 citations recorded by the source corpus.
Subfields: Infectious Diseases, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/2f3367b17a0ee5cde5e86f7c6c13f96eaa899924
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In Infectious Diseases
In Molecular Biology
Citations in the corpus
- Neutrophil Extracellular Traps Contain Calprotectin, a Cytosolic Protein Complex Involved in Host Defense against Candida albicans (2009, background): “The ability to reversibly switch between different morphologies upon external stimuli appears to be essential for the virulence of C. albicans [22,23].”
- Systematic screens of a Candida albicans homozygous deletion library decouple morphogenetic switching and pathogenicity (2010, background): “(4) ORFs encoding She3-associated mRNAs, n=5120.”
- Candida albicans pathogenicity mechanisms (2013, background): “Further morphologies include white and opaque cells, formed during switching, and chlamydospores, which are thick-walled spore-like structures.(17) While yeast and true hyphae are regularly observed during infection and have distinct functions (as discussed below), the role of pseudohyphae and switc”
- Growth of Candida albicans hyphae (2011, background): “albicans is its ability to grow either as a unicellular budding yeast or in filamentous pseudohyphal and hyphal form”