Macrophage activation switching: an asset for the resolution of inflammation

By F. Porcheray, S. Viaud, A. Rimaniol, C. Léone, B. Samah, N. Dereuddre-Bosquet, D. Dormont, G. Gras. Clinical and Experimental Immunology, 2005

903 citations recorded by the source corpus.

Subfields: Immunology, Neurology

View Paper: https://www.semanticscholar.org/paper/2e9516e56e528e1bafdca45b24b4bbec3ef283f1

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Citations in the corpus

Macrophage Dysfunction Impairs Resolution of Inflammation in the Wounds of Diabetic Mice (2010, background): “In response to cues from the microenvironment, pro-inflammatory activated M1 macrophages may switch to M2 [65].”
Macrophage Polarization in Bacterial Infections (2008, background): “Recently, macrophage activation has been shown to be plastic, rapid, and fully reversible, suggesting that macrophage populations are dynamic and may first take part in inflammation and then participate in its resolution (11).”
Understanding the Mysterious M2 Macrophage through Activation Markers and Effector Mechanisms (2015, background): “Surprisingly IL-4 represses or does not affect its expression [27, 90].”
Macrophage phenotypes in atherosclerosis (2014, background): “In contrast to the M1 and M2 polarization induced by GM-CSF and M-CSF or Th1 and Th2 cytokines, which are reversible processes (65), a novel human macrophage phenotype induced by the platelet-derived chemokine CXCL4 was reported to be irreversible (66).”
Macrophages and Tissue Injury: Agents of Defense or Destruction? (2011, background): “Macrophage activation is in fact a dynamic process; the same cells may initially take part in proinflammatory and cytotoxic reactions and later participate in the resolution of inflammation and wound healing (5, 8, 9).”
Macrophages in skin injury and repair. (2011, positive): “It has furthermore been suggested that M2 macrophages derived from M1 macrophages contribute to the resolution of inflammation and the wound healing process (Goerdt and Orfanos 1999; Porcheray et al. 2005).”
Inflammatory monocytes recruited after skeletal muscle injury switch into antiinflammatory macrophages to support myogenesis (2007, background): “extent these distinct activation states exist in vivo and whether MP fate is determined for ever or whether it remains constantly malleable (2), although several in vitro studies have shown MP adaptation to changing microenvironment (4,6-8).”
Mesenchymal stem cell-educated macrophages: a novel type of alternatively activated macrophages (2009, background): “To test our hypothesis that MSCs induce anti-inflammatory phenotype in macrophages, we examined cell surface expression of CD206, a well known marker for anti-inflammatory macrophages [27].”