Increased adipose tissue in male and female estrogen receptor-alpha knockout mice.

By P. Heine, J. Taylor, G. Iwamoto, D. Lubahn, P. Cooke. Proceedings of the National Academy of Sciences of the United States of America, 2000

594 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/2cea5afc503444cc6c9bd0be64894d81a896cede

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

1. André Tchernof
2. J. Despres
3. G. Steinberg
4. Bruce E. Kemp
5. S. Grundy
6. I. Graham

Citations in the corpus

• AMPK in Health and Disease. (2009, background): “Since the constitutively active AMPK fragment 1 (1-312) is not activated by the PT1 compound, it is proposed that PT1 acts by reversing the autoinhibition of the AIS (313-335).”
• Ten Putative Contributors to the Obesity Epidemic (2009, unknown): “The estrogen receptor-α knock out (αERKO) mouse model further argues for the role of estrogen in regulating WAT (Heine et al., 2000).”
• Pathophysiology of human visceral obesity: an update. (2013, background): “Interestingly, deletion of the estrogen receptor in male and female mice is associated with increased adiposity independent of food intake (216).”