Tumor angiogenesis: past, present and the near future.
By R. Kerbel. Carcinogenesis, 2000
1,027 citations recorded by the source corpus.
Subfields: Cancer Research, Molecular Biology, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/2bee8be059000b5a7370f98b8581cbf4954da649
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Citations in the corpus
- Colony-Stimulating Factor 1 Promotes Progression of Mammary Tumors to Malignancy (2001, background): “On the other hand, macrophages have also been found to promote tumor angiogenesis (14) and to secrete a wide range of growth factors which may promote tumor growth (15).”
- Continuous low-dose therapy with vinblastine and VEGF receptor-2 antibody induces sustained tumor regression without overt toxicity. (2000, background): “Intriguingly, this list may include many different conventional cytotoxic chemotherapeutic drugs (2), radiation (13), and hormonal ablation therapies (14, 15).”
- The role of disturbed pH dynamics and the Na+/H+ exchanger in metastasis (2005, background): “Indeed, both progression and patient mortality are correlated with microvascular density, which is a measure of the development of the metabolic microenvironmen”
- Vascular Endothelial Growth Factor and Angiogenesis (2004, background): “The formation of new blood vessels provides a mechanism by which an in situ tumor lesion can circumvent the critical limitations of the oxygen diffusion distance and restrictions on nutrient exchange (Folkman, 1995; Carmeliet, 2000; Carmeliet and Jain, 2000; Kerbel, 2000).”
- Angiogenesis in cancer and other diseases (2000, background): “These include metabolic stress (for example, low p O 2 , low pH or hypoglycaemia), mechanical stress (for example, pressure generated by proliferating cells), immune/inflammatory response (for example, immune/inflammatory cells that have infiltrated the tissue), and genetic mutations (for examp”