Human Keratinocytes That Express hTERT and Also Bypass a p16INK4a-Enforced Mechanism That Limits Life Span Become Immortal yet Retain Normal Growth and Differentiation Characteristics
By M. Dickson, William C. Hahn, Y. Ino, V. Ronfard, Jenny Y. Wu, R. Weinberg, D. Louis, Frederick P. Li, J. Rheinwald. Molecular and Cellular Biology, 2000
1,119 citations recorded by the source corpus.
Subfields: Cell Biology, Dermatology, Physiology
View Paper: https://www.semanticscholar.org/paper/2b406733e16479abafa01ab3257b55a0a41a157c
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Citations in the corpus
- Comparative biology of mouse versus human cells: modelling human cancer in mice (2003, background): “cgi?l=7015">TERT ; the catalytic subunit of human telomerase) into these cells enables them to undergo immortalizatio”
- Als3 Is a Candida albicans Invasin That Binds to Cadherins and Induces Endocytosis by Host Cells (2007, background): “Rheinwald (Harvard University, Cambridge, Massachusetts, United States) [30].”
- The molecular biology of head and neck cancer (2011, background): “Moreover, in most in vitro models, TERT seemed to be an important facto”
- Hypoxia-inducible factor-1-dependent regulation of the multidrug resistance (MDR1) gene. (2002, background): “grown and cultured as described previously (17) and were a kind gift from Dr.”