Prognostic Value of Coronary Vascular Endothelial Dysfunction

By J. Halcox, W. Schenke, G. Zalos, R. Mincemoyer, A. Prasad, M. Waclawiw, K. R. Nour, A. Quyyumi. Circulation, 2002

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The relationship between shear stress and flow‐mediated dilatation: implications for the assessment of endothelial function (2005, background): “Many studies have in fact noted an inverse relationship between vessel size and FMD (Celermajer et al. 1992; Anderson et al. 1995; Herrington et al. 2001) but the mechanism (that smaller vessels experience a larger stimulus) has not been reported.”
Contemporary Reviews in Cardiovascular Medicine The Assessment of Endothelial Function From Research Into Clinical Practice (2012, neutral): “ments cardiovasculaires, y compris chez les patients indemnes de coronaropathie.(106,107) Cela étant, il ne peut être envisagé de pratiquer des mesures intrusives en prévention primaire ; de”
Oxidant Mechanisms in Renal Injury and Disease (2016, neutral): “If the reduction in nitric oxide persists, endothelial dysfunction ensues and is potentially followed by the development of cardiovascular and renal diseases (71, 101, 104, 133).”
Coronary microvascular reactivity to adenosine predicts adverse outcome in women evaluated for suspected ischemia results from the National Heart, Lung and Blood Institute WISE (Women's Ischemia Syndrome Evaluation) study. (2010, background): “Other studies indicate that positron emission tomography easures of absolute myocardial blood flow (MBF) and FR or myocardial perfusion reserve (1,2,5,7,10,14) might e abnormal in individuals with risk factors without apparnt CAD.”