Cytosolic FoxO1 is essential for the induction of autophagy and tumour suppressor activity
By Ying Zhao, Jing Yang, Wenjuan Liao, Xiangyu Liu, Hui Zhang, Shan Wang, Donglai Wang, Jingnan Feng, Li Yu, Wei-Guo Zhu. Nature Cell Biology, 2010
614 citations recorded by the source corpus.
Subfields: Epidemiology, Geriatrics and Gerontology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/2939796d0a0278b4feeaeebe06671b6d52394fb7
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- Cristina Sánchez-Ramos
- Ignacio Prieto-Arroyo
- L. Klotz
- M. Monsalve
- P. Urbánek
- H. Steinbrenner
Citations in the corpus
- Redox regulation of FoxO transcription factors (2015, background): “Acetylated FoxO1a then induced the autophagic process through interaction with Atg7 (ubiquitin-like modifier-activating enzyme 7), a key regulator in the formation of the autophagosome [215].”
- Autophagy in malignant transformation and cancer progression (2015, neutral): “As an increasing number of activities is being ascribed to these and other autophagy mediators (Cosse et al , 2010; Lee et al , 2012; Liu et al , 2012a; Moscat & Diaz-Meco, 2012; Zhao et al , 2012; Maskey et al , 2013; Elgendy et al , 2014), it remains possible that other, autophagy-independent func”