The Haber-Weiss reaction and mechanisms of toxicity.
By J. Kehrer. Toxicology, 2000
1,347 citations recorded by the source corpus.
Subfields: Inorganic Chemistry, Molecular Biology, Organic Chemistry
View Paper: https://www.semanticscholar.org/paper/291f59154daf479480e6e6ea9c9754091531151f
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In Inorganic Chemistry
In Molecular Biology
In Organic Chemistry
Citations in the corpus
- Cadmium stress: an oxidative challenge (2010, neutral): “Whereas the production of radicals by mitochondria in healthy tissues is rather low, ROS production substantially increases in the presence of xenobiotics (Kehrer 2000).”
- Reactive oxygen species as signals that modulate plant stress responses and programmed cell death (2006, methodology): “(4) O 2 is formed mainly by electron leakage from Fe-S centers of photosystem I or reduced ferredoxin to O2 (Mehler reaction), which is then quickly metabolized to H2O2 by SOD.”
- The role of oxidative stress in ambient particulate matter-induced lung diseases and its implications in the toxicity of engineered nanoparticles. (2008, background): “Fe, Ni, Cu, Co, and Cr) may play an important role in this effect [29–31].”
- The role of phenolic compounds in plant resistance (2016, background): “The conjunction of both equations gives Haber-Weiss reaction [19]:”
- Redox Regulation of Cell Survival (2008, neutral): “In the presence of Fe(II) and Fe(III), HO can be generated through the Fenton reaction or Haber–Weiss reaction (151).”
- Acquisition and Homeostasis of Iron in Higher Plants and Their Probable Role in Abiotic Stress Tolerance (2018, background): “Excess free Fe ions like Fe2+ and Fe3+ can cause ROS generation by participating in the Fenton reaction (Fenton, 1894; Haber and Weiss, 1934; Kehrer, 2000; Mai and Bauer, 2016) and leading to oxidative stress (Mai and Bauer, 2016).”
- The Chemistry of Reactive Oxygen Species (ROS) Revisited: Outlining Their Role in Biological Macromolecules (DNA, Lipids and Proteins) and Induced Pathologies (2021, background): “The hydroxyl radical •OH (Figure 3A–C), the most powerful ROS oxidant, is formed during the Haber–Weiss reaction [8], by the Fenton reaction [9] or by decomposition of peroxynitrite [10], and has a very short half-life (10−9 s) and high reactivity.”
- The effects of oxidative stress on female reproduction: a review (2012, methodology): “Placental OS can be detected through increased serum concentrations of ROS such as H2O2 [194], or lipid peroxidation markers [195] such as MDA [179,195-197] and thiobarbituric acid reactive substances (TBARS) [179,194].”
- ROS-mediated abiotic stress-induced programmed cell death in plants (2015, background): “Chloroplasts, as well as mitochondria, send signals to the nucleus, a process known as retrograde signaling (Suzuki et al., 2012), and in this way they may influence the expression of numerous genes in accordance with their redox condition, including triggers of PCD.”