Splenic nerve is required for cholinergic antiinflammatory pathway control of TNF in endotoxemia

By M. Rosas‐Ballina, M. Ochani, W. Parrish, K. Ochani, Y. Harris, J. Huston, S. Chavan, Kevin J. Tracey. Proceedings of the National Academy of Sciences of the United States of America, 2008

706 citations recorded by the source corpus.

Subfields: Molecular Biology, Neurology, Psychiatry and Mental health

View Paper: https://www.semanticscholar.org/paper/251b3cb3dd018374522484357e56ff5188ec20ed

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Molecular Biology

1. C. Deutschman
2. Bruno Bonaz
3. C. Bernstein
4. Kevin J. Tracey
5. Bruce S. McEwen
6. Peter J. Gianaros

In Neurology

1. C. Deutschman
2. Bruno Bonaz
3. C. Bernstein
4. Kevin J. Tracey
5. Bruce S. McEwen
6. Peter J. Gianaros

In Psychiatry and Mental health

1. C. Bernstein
2. Bruno Bonaz

Citations in the corpus

• Brain-gut interactions in inflammatory bowel disease. (2013, background): “The VN also indirectly modulates immune activity of the spleen through its connections with the splenic sympathetic nerve (29).”
• Sepsis: current dogma and new perspectives. (2014, background): “…or splenic nerves, genetically ablating a7nAChR, or pharmacologically inhibiting signals along this pathway all enhance cytokine release and the increase the severity of of lethal shock and tissue injury (Bernik et al., 2002; Borovikova et al., 2000; Rosas-Ballina et al., 2008; Wang et al., 2003).”