Type 1 diabetes: new perspectives on disease pathogenesis and treatment.
By Mark Atkinson, G. Eisenbarth. The Lancet, 2001
1,575 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Genetics, Surgery
View Paper: https://www.semanticscholar.org/paper/24a19912850c7e8fc3b28ccec20c2cef16def5c1
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In Endocrinology, Diabetes and Metabolism
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Citations in the corpus
- The Role of Inflammation in Diabetes: Current Concepts and Future Perspectives (2018, background): “The first theories about predisposition support that environmental trigger factors in early life, such as infections, nutrition and chemicals that are able to activate self-targeting immune cascades, remain applicable even though the initial event is still unclear.(29,30)”
- Staging Presymptomatic Type 1 Diabetes: A Scientific Statement of JDRF, the Endocrine Society, and the American Diabetes Association (2015, background): “In children and adults, the rate of progression fromonset ofb-cell autoimmunity to glucose intolerance and then to symptomatic disease is variable, lasting from months to decades (2,3).”
- Type 1 diabetes (2013, methodology): “IAA insulin autoantibodies, ICAs islet cell cytoplasmic autoantibodies, GADAs glutamic acid decarboxylase autoantibodies (adapted from Atkinson and Eisenbarth [ 1 ] , Eisenbarth [ 6 ] , and Devendra [ 7 ] ).”
- Guidelines and recommendations for laboratory analysis in the diagnosis and management of diabetes mellitus. (2002, background): “No acceptable therapy has been demonstrated to prolong the survival of islet cells once diabetes has been diagnosed or to prevent the clinical onset of diabetes in islet cell autoantibody–positive individuals (279).”
- Guidelines and Recommendations for Laboratory Analysis in the Diagnosis and Management of Diabetes Mellitus (2002, background): “The following suggestions have been proposed by Atkinson and Eisenbarth (204) as a rational approach to the use of autoantibodies in diabetes: (a) antibody assays should have specificity 99%; (b) proficiency testing should be documented; (c) multiple autoantibodies should be assayed; and (d) sequent”
- Type 1 diabetes: etiology, immunology, and therapeutic strategies. (2011, background): “T1D is a chronic autoimmune disorder that precipitates in genetically susceptible individuals by environmental factors (24).”
- Effects of oral insulin in relatives of patients with type 1 diabetes: The Diabetes Prevention Trial--Type 1. (2005, background): “Type 1 diabetes is a consequence of immune-mediated destruction of insulinsecreting pancreatic islet -cells (4).”