Regulation of Toll/IL-1-receptor-mediated gene expression by the inducible nuclear protein IκBζ
By Masahiro Yamamoto, S. Yamazaki, Satoshi Uematsu, Shintaro Sato, H. Hemmi, K. Hoshino, T. Kaisho, H. Kuwata, O. Takeuchi, K. Takeshige and 7 more. Nature, 2004
487 citations recorded by the source corpus.
Subfields: Cancer Research, Immunology
View Paper: https://www.semanticscholar.org/paper/223b69ae3b6bfce72ec9480e914563fc90f92b6e
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In Cancer Research
- S. Ghosh
- M. Hayden
- Shizuo Akira
- Satoshi Uematsu
- O. Takeuchi
- Alexander Hoffmann
- Kenta Nakai
- Daron M. Standley
In Immunology
Citations in the corpus
- New regulators of NF-κB in inflammation (2008, background): “However, rather than providing a negative feedback effect, such as that provided by typical IκB proteins, IκBζ can positively regulate certain NF-κB-dependent genes in response to TLR4 or IL-1R stimulatio”
- The Jmjd3-Irf4 axis regulates M2 macrophage polarization and host responses against helminth infection (2010, background): “IκBζ, an IκB family member, positively regulates certain genes such as II6 by interacting with NF-κBp50 or inducing histone modificatio”