Cardiovascular psychophysiology
By S. Knardahl. Annals medicus, 2000
665 citations recorded by the source corpus.
Subfields: Applied Psychology, Behavioral Neuroscience, Cardiology and Cardiovascular Medicine
View Paper: https://www.semanticscholar.org/paper/1f6cb6f0ac3a6a5a6d3a6ca8a60fe4d30ebb7ce8
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Applied Psychology
- Barbara L. Fredrickson
- Christian E. Waugh
- Michael Richter
- Jason W. Osborne
- Sylvain Laborde
- D. Noll
- H. Koenig
- Martin A. Lindquist
In Behavioral Neuroscience
- Sylvain Laborde
- D. Noll
- H. Koenig
- Michael Richter
- Julian F. Thayer
- Martin A. Lindquist
- Barbara L. Fredrickson
- Stephan F. Taylor
In Cardiology and Cardiovascular Medicine
- F. Shaffer
- Sylvain Laborde
- E. Mosley
- Julian F. Thayer
- Barbara L. Fredrickson
- Rollin McCraty
- Stephen W. Porges
- Tor D. Wager
Citations in the corpus
- Linking Stereotype Threat and Anxiety (2007, background): “Blood vessels tend to constrict when an individual is coping with aversive stimuli, and is most clearly a sympathetic nervous system reaction (Bradley et al., 1997; Brownly et al., 2000).”
- Brain mediators of cardiovascular responses to social threat, Part I: Reciprocal dorsal and ventral sub-regions of the medial prefrontal cortex and heart-rate reactivity (2009, methodology): “sympathetic system affects HR with an approximately 2 sec longer response latency than the parasympathetic system, which can exert effects within hundreds of milliseconds (Berntson et al., 2007).”
- A healthy heart is not a metronome: an integrative review of the heart's anatomy and heart rate variability (2014, background): “In contrast, Berntson et al. (2007) recommend a minimum sampling rate of 500–1000 Hz.”
- Effort Mobilization when the Self is Involved: Some Lessons from the Cardiovascular System (2010, background): “HR is determined by both sympathetic and parasympathetic arousal and thus should only respond to effort mobilization when the sympathetic impact is stronger, which is not always the case (Berntson, Cacioppo, & Quigley, 1993; Brownley et al., 2000; Obrist, 1981).”
- Heart Rate Variability and Cardiac Vagal Tone in Psychophysiological Research – Recommendations for Experiment Planning, Data Analysis, and Data Reporting (2017, background): “More conservative guidelines advise between 500 and 1000 Hz (Riniolo and Porges, 1997; Berntson et al., 2007), which may be particularly relevant in case of lower amplitude RSA (Riniolo and Porges, 1997).”
- Religion, Spirituality, and Health: The Research and Clinical Implications (2012, background): “The word “hypertension” itself suggests a relationship with stress or tension, and high blood pressure has been linked to greater psychosocial stress [379–381].”
- Don't fear ‘fear conditioning’: Methodological considerations for the design and analysis of studies on human fear acquisition, extinction, and return of fear (2017, neutral): “Both conitioned HR decelerations and conditioned HR accelerations have een observed with the former presumably indicating an orienting esponse to the signal value of the CS and the latter a defensive esponse and, thus, reflecting learned fear (Hamm et al., 1993). ccordingly, conditioned HR decelerat”
- The polyvagal perspective. (2007, background): “…psychophysiology: one, proposed by Lacey (1967), hypothesized that baroreceptors were involved in mediating the relation between sensory thresholds and short latency directional heart rate changes; and the other, proposed by Obrist (1981), linked heart rate levels to metabolic demands.”
- Stress and health: psychological, behavioral, and biological determinants. (2005, background): “Chronically elevated blood pressure forces the heart to work harder, which leads to hypertrophy of the left ventricle (Brownley et al. 2000).”
- Challenge and Threat During Social Interactions With White and Black Men (2002, background): “As Brownley, Hurwitz, and Schneiderman (2000) describe, “an increase in afterload (i.e., the cumulative effects of systemic vascular resistance, blood viscosity and volume, and vascular compliance on resistance to flow) tends to limit or reduce stroke volume” (p. 228).”