Gene-expression profiles in hereditary breast cancer.

By J. Sudbø, A. Reith, O. Lingjaerde. New England Journal of Medicine, 2001

907 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/1d5b3054f267584dc5a730e9e3754b682d7888b8

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Citations in the corpus

A gene-expression signature as a predictor of survival in breast cancer. (2002, unknown): “7 In addition, microarray analysis has been used to distinguish cancers associated with BRCA1 or BRCA2 mutations 8,9 and to determine estrogen-receptor status 6,9,10 and lymph-node status.”
BRCA1 and BRCA2: 1994 and beyond (2004, background): “fcgi?db=gene&cmd=Retrieve&dopt=Graphics&list_uids=595">cyclin D1 seems to be a useful marker for BRCA2 -related breast cance”
BioArray Software Environment (BASE): a platform for comprehensive management and analysis of microarray data (2002, background): “Whereas microarrays have shed light on many biological processes and disease states, for us [7-10] and others, a significant bottle-neck remains the analysis of hybridization data in the context of biomaterial and reporter annotations.”
Prediction of BRCA1 Status in Patients with Breast Cancer Using Estrogen Receptor and Basal Phenotype (2005, background): “3), and recently, this has been confirmed on expression analysis (9, 13 ).”
Hallmarks of 'BRCAness' in sporadic cancers (2004, background): “However, gene-expression microarray analysis does indicate the distinct nature of familial- BRCA1 ,familial- BRCA2 and unselected sporadic breast tumour”
Coexpression analysis of human genes across many microarray data sets. (2004, unknown): “2001) 58 2756 24781 (Unpublished, GSE470) 85 8243 241088 (Hedenfalk et al. 2001) 22 2253 1265 (Unpublished, GSE474) 10 5418 7941 (Hedenfalk et al.”
The fundamental role of epigenetic events in cancer (2002, background): “Furthermore, microarray studies indicate that the overall gene-expression profiles of sporadic breast cancers with hypermethylated BRCA1 are identical to those of the familial cancers in which BRCA1 is mutated and are distinct from those of other breast-cancer type”
A Factor Model Approach to Multiple Testing Under Dependence (2009, methodology): “The above factor analysis modeling of the conditional variance is hereafter applied on well-known real microarray data which were primarily analyzed by Hedenfalk et al. (2001) in order to compare expressions of three types of breast cancer tumor tissues: BRCA1, BRCA2, and Sporadic.”
Classification and diagnostic prediction of cancers using gene expression profiling and artificial neural networks (2001, methodology): “t(11;22)(q24;q12) in EWS, and the PAX3-FKHR; t(2;13)(q35;q14) in alveolar rhabdomyosarcoma(1) (ARMS).”
Meta-analysis of microarrays: interstudy validation of gene expression profiles reveals pathway dysregulation in prostate cancer. (2002, methodology): “Ps were assigned to each gene with permutation t tests (4, 12) and, thus, represent the likelihood that expression values of a gene were associated with samples at random.”
Capturing Heterogeneity in Gene Expression Studies by Surrogate Variable Analysis (2007, methodology): “The second dataset consists of gene expression for 3,226 genes in seven BRCA1 and eight BRCA2 mutation–positive tumor samples [22]; several genes with apparent outliers were removed as described [23] for a total of 3,170 genes.”
Race, breast cancer subtypes, and survival in the Carolina Breast Cancer Study. (2006, unknown): “Well-/moderately differentiated 174 (35) 18 (18) 10 (30) 106 (42) 34 (44) 6 (19) .”
Epigenetic gene silencing in cancer: the DNA hypermethylome. (2007, background): “We can examine a number of aspects: (a) whether the reintroduction of the gene in a deficient cancer cell line reduces colony formation, or inhibits xenograft growth in nude mice; (b) whether the hypermethylation of that gene is associated with a particular molecular or clinical phenotype, as is the”
Estimating the Null and the Proportion of Nonnull Effects in Large-Scale Multiple Comparisons (2006, background): “Among the 496 genes identified as being differentially expressed by the local FDR procedures, 17 of them have been discovered in the study by Hedenfalk et al. (2001).”
Bayesians, Frequentists, and Scientists (2005, background): “Expression Data for the First of 3,226 Genes, Microarray Study of Breast Cancer (Hedenfalk et al. 2001).”