Keeping pain out of mind: the role of the dorsolateral prefrontal cortex in pain modulation.
By Jürgen Lorenz, Satoshi Minoshima, K. Casey. Brain : a journal of neurology, 2003
936 citations recorded by the source corpus.
Subfields: Cognitive Neuroscience, Physiology
View Paper: https://www.semanticscholar.org/paper/1d368079c715e59f433d72889c71a1540161f60f
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In Cognitive Neuroscience
- Irene Tracey
- Jürgen Lorenz
- Niels Birbaumer
- Christian Büchel
- Rolf-Detlef Treede
- P. Mantyh
- R. Baron
- Richard S. J. Frackowiak
In Physiology
Citations in the corpus
- Expectancy and belief modulate the neuronal substrates of pain treated by acupuncture (2005, background): “Lorenz et al. proposed that the DLPFC exerts an active control on pain perception by modulating cortico-cortical and cortico-subcortical pathways (Lorenz et al., 2003).”
- Activation of the opioidergic descending pain control system underlies placebo analgesia. (2009, background): “Upon painful stimulation, treatment expectations regarding the analgesic efficacy of the placebo cream need to be ‘‘kept in mind’’ and pain needs to be ‘‘kept out of mind’’ (Bunge et al., 2001; Lorenz et al., 2003).”
- The cerebral signature for pain perception and its modulation. (2007, result): “…Frontal, and Insular Cortex in Chronic Pain It is clear from these few studies described and others in the literature (Apkarian et al., 2001, 2005; Lorenz et al., 2003; Phillips et al., 2003; Witting et al., 2006), that rostral anterior insula and pronounced PFC activation are consistently found…”
- Quantitative sensory testing in the German Research Network on Neuropathic Pain (DFNS): Somatosensory abnormalities in 1236 patients with different neuropathic pain syndromes (2010, background): “8%), the latter suggesting a contribution of central mechanisms [19,35].”
- Distraction modulates connectivity of the cingulo‐frontal cortex and the midbrain during pain—an fMRI analysis (2004, background): “demonstrated a functional relationship of the dorsolateral prefrontal cortex (DLPFC) with thalamus and midbrain and proposed that the DLPFC exerts active control on pain perception by modulating corticosubcortical and corticocortical pathways (Lorenz et al., 2003).”