Human papillomavirus and oral cancer: the International Agency for Research on Cancer multicenter study.

By R. Herrero, X. Castellsagué, M. Pawlita, J. Lissowska, F. Kee, P. Balaram, T. Rajkumar, H. Sridhar, B. Rose, J. Pintos and 13 more. Journal of the National Cancer Institute, 2003

1,288 citations recorded by the source corpus.

Subfields: Epidemiology, Otorhinolaryngology, Periodontics

View Paper: https://www.semanticscholar.org/paper/1c0488e332636ddacd73327935a4772d27b8800c

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

In Otorhinolaryngology

Citations in the corpus

Improved survival of patients with human papillomavirus-positive head and neck squamous cell carcinoma in a prospective clinical trial. (2008, neutral): “6 10 – 32 ounces 9 (24) 10 (17) >32 ounces 6 (16) 13 (22) Unknown 2 (5) 5 (9) Smoking history, No.”
Pathogenesis of Human Papillomaviruses in Differentiating Epithelia (2004, background): “pharynx contain DNA from these HPV types (61).”
Tongue and tonsil carcinoma (2005, neutral): “20–44 871 (39) 711 (31) 355 (16) 189 (8) 136 (6) 45 7216 (23) 8468 (27) 6697 (21) 5383 (17) 3838 (12)”
Interaction between tobacco and alcohol use and the risk of head and neck cancer: pooled analysis in the INHANCE consortium (2009, methodology): “Studies of HPV in North America provide some preliminary evidence that this may be true (27, 28), in contrast to a study including cases mostly from Europe (12).”
The global health burden of infection‐associated cancers in the year 2002 (2006, methodology): “The largest study so far is a multicentre case-control study in 9 countries, including more than 1,600 cases of cancers of the mouth and oropharynx and 1,700 controls.(38) HPV DNA was detected in tumour specimens (cases) by PCR, and presence of antibodies against HPV 16 L1 and HPV 16 E6 and E7 was t”
Human Papillomavirus Types in Head and Neck Squamous Cell Carcinomas Worldwide: A Systematic Review (2005, background): “Over the past 15 years, human papillomavirus (HPV), the necessary cause of cancer of the cervix (3, 4), has also been etiologically linked with a subset of HNSCCs (1, 5-8).”
Worldwide trends in incidence rates for oral cavity and oropharyngeal cancers. (2013, background): “For example, contemporary estimates indicate that approximately 60% to 70% of OPCs are caused by HPV infection in the United States,(4,22,30) compared with less than 10% in less economically developed regions.(29,31) Similarly, in a comprehensive review of the global burden of infection-associated c”
Human papillomavirus molecular biology and disease association (2015, background): “HPV prevalence is significantly higher in oropharynx SCC than in the oral cavity with the tonsil having higher prevalence than other anatomic sites [58].”