The molecular basis of lung morphogenesis.
By D. Warburton, M. Schwarz, D. Tefft, G. Flores‐Delgado, K. Anderson, W. Cardoso. Mechanisms of Development, 2000
738 citations recorded by the source corpus.
Subfields: Molecular Biology, Pulmonary and Respiratory Medicine, Surgery
View Paper: https://www.semanticscholar.org/paper/18860099f5ce4338e0988e7bbb3f9ebe89e96e8f
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In Molecular Biology
In Pulmonary and Respiratory Medicine
In Surgery
Citations in the corpus
- Meta-analyses of genome-wide association studies identify multiple novel loci associated with pulmonary function (2009, background): “The NPNT SNP rs10516529 is located in a binding site for the transcription factor POU6F1 (also known as mPOU homeobox protein), which is known to be expressed in adult lungs and hypothesized to play a role in lung developmen”
- Pulmonary neuroendocrine (carcinoid) tumors: European Neuroendocrine Tumor Society expert consensus and recommendations for best practice for typical and atypical pulmonary carcinoids. (2015, background): “PCs are tumors with NE morphology and differentiation, which are deemed to derive from, or differentiate toward, the mature NE cells of the pulmonary diffuse NE system [60–62].”
- Placental development: Lessons from mouse mutants (2001, background): “In the lung and kidney, for example, the reciprocal interactions between the organ's epithelium and mesenchyme that lead to branching morphogenesis are thought to secondarily regulate the elaboration of the vasculatur”
- Familial primary pulmonary hypertension (gene PPH1) is caused by mutations in the bone morphogenetic protein receptor-II gene. (2000, background): “…protein (BMP) receptor type II (BMPR-II [gene BMPR2, also known as “T-ALK,” “CL4-1,” and “BRK-3”]), a member of the transforming growth factor b (TGF-b)–receptor superfamily, was suggested on the basis of the role of the BMP-signaling pathway in lung morphogenesis (Warburton et al. 2000).”